Dysmetabolic Nephropathy

Primary and secondary dysmetabolic nephropathy is allocated depending on the causes.

In the first case, the disease is hereditary and is characterized by development in the early stages of urolithiasis and progressive course. There is also a development of chronic kidney failure.

Secondary dysmetabolic nephropathy can be associated with excessive intake of any substances in the body or with diseases of the gastrointestinal tract. The most common secondary dismetabolic nephropathy.

Diseases in the form of oxalate-calcium nephropathy are more common in children. The cause of the occurrence is, as a rule, a violation of the exchange of oxalates and calcium. The causes of oxalate formation can be intestinal anastomoses, ulcerative colitis, Crohn's disease, and excessive intake of oxalates with food. Oxalatum dysmetabolic nephropathy in children in most cases is hereditary. An important role, besides genetic factors, is played by an unfavorable ecological situation, unbalanced nutrition and stresses. The first signs can appear even in newborns, but are more often detected at the age of 6-7 years. For children with this disease are characterized by vegeto-vascular dystonia, obesity, allergies. The overall development of the child with this disease does not suffer. Progression of nephropathy can lead to the development of urolithiasis, and with the layering of bacterial infection - to inflammation of the kidneys.

Phosphate dismetabolic nephropathy is a sign of impaired phosphoric-calcium metabolism. The main cause of its occurrence are chronic infections of the urinary system. Urine nephropathy is a violation of uric acid metabolism. A characteristic sign in this disease is urine of brick color.

Treatment of dismetabolic nephropathy consists in the application of specific methods of therapy, diet and proper drinking regimen, in the overall normalization of the way of life. A universal method of treating nephropathy is the use of a large volume of fluid, as this reduces the concentration of substances soluble in urine. The intake of fluid before bed helps to increase the volume of nocturnal urination. Take preferably plain or mineral water. The goal of the diet is to reduce the salt load on the kidneys. Specific therapy is directed at normalization of metabolic processes, elimination of salts and prevention of crystal formation processes.

In the treatment of oxalate nephropathy patients are assigned a cabbage-potato diet. The diet excludes strong meat broths, chillies, chocolate, cocoa, sorrel and spinach, carrots, beets and cranberries. Recommended use of pears, prunes and dried apricots. Medicinal therapy consists in the intake of antioxidants and membranotropic drugs. Treatment is long-term. It is prescribed vitamin B6 (pyridoxine) monthly course, as well as vitamin A, which normalizes the interaction of lipids and proteins of the cell membrane. Vitamin E (tocopherol acetate) is a powerful antioxidant and also strengthens the lipid-protein bonds of cell membranes. Used also as membrane stabilizers, xidiphon and dimephosphon. In the treatment of crystalluria, the efficacy of cystone and magnesium oxide is shown.

From the diet should be excluded rich in purine-based foods, such as kidneys, liver, nuts, beans, peas. It is necessary to use mainly products of vegetable and milk origin. Good results are obtained by the intake of a large number of liquids, including decoctions of such herbs as horsetail, clover, sporich. Mineral water is slightly alkaline slightly mineralized.